For research and educational purposes only. Not medical advice.
Apelin-13 Reference
Educational, not medical advice reference for Apelin-13: Metabolic; regulatory status, evidence posture, source review, and schedule notes. Also known as [Py…
Reference summary
Maguire 2009 (Hypertension) identified [Pyr1]apelin-13 as the predominant apelin isoform in the human heart. Japp 2010 (Circulation) showed acute IV infusion (30 to 300 nmol/min) increased cardiac index and reduced mean arterial pressure and peripheral vascular resistance in healthy volunteers and chronic heart failure patients. Brash 2018 (JACC Basic Transl Sci) showed acute IV infusion reduced pulmonary vascular resistance and increased cardiac output in pulmonary arterial hypertension.
- Categories
- Metabolic
- Aliases
- [Pyr1]apelin-13, pyr-Apelin-13, Pyroglutamated apelin-13, APJ receptor agonist
- Evidence posture
- human - Foundational small human IV-infusion data exist, but native [Pyr1]apelin-13 has a very short half-life and there is no FDA-approved apelin drug. Modern programs use long-acting analogs. Not a research-vial peptide with a validated subcutaneous dosing convention.
- Regulatory status
- No FDA-approved apelin drug label. Apelin / [Pyr1]apelin-13 is an endogenous APJ-receptor peptide. There is no active standalone drug program in the United States; therapeutic development uses long-acting analogs because the native peptide has a very short half-life.
- Content review status
- investigational verified
Selected public sources
- PubMed: Japp 2010 - Acute cardiovascular effects of apelin in humans (Circulation)
- PubMed: Maguire 2009 - [Pyr1]apelin-13 identified as the predominant apelin isoform in the human heart (Hypertension)
- PubMed: Brash 2018 - Short-term hemodynamic effects of apelin in patients with pulmonary arterial hypertension (JACC Basic Transl Sci)